Korean Journal of Gastrointestinal Endoscopy 2007;34(4): 205-209.
십이지장 협착으로 인한 위출구폐쇄에 의해 악화된 식도칸디다증 및 효모위석 발생 1예
송정훈·이은빈·박남선·강병국·진대호·안태홍·한윤주·이규철·이형석
서울적십자병원 내과
A Case of Aggravated Esophageal Candidiasis and Gastric Yeast Bezoar Formation following Gastric Outlet Obstruction due to Duodenal Stenosis
Jung Hoon Song, M.D., Eun Bin Lee, M.D., Nam Seon Park, M.D., Byung Kook Kang, M.D., Dae Ho Jin, M.D., Tae Hong Ahn, M.D., Yoon Ju Han, M.D., Kyu Chel Lee, M.D. and Hyung Suk Lee, M.D.
Department of Internal Medicine, Seoul Red Cross Hospital, Seoul, Korea
Abstract
Esophageal candidiasis is the most common disease among all candida infections of the gastrointestinal tract, and generally develops in immunocompromised patients. The prevalence of esophageal candidiasis has increased in patients undergoing antibiotic therapy, diabetes, adrenal dysfunction, alcohol intoxication, old age, esophageal injury, esophageal stasis, gastric surgery, and acid suppressive therapy. However, the overall prevalence is not higher than that of immunocompromised patients. Gastric candidiasis is uncommon because of the strong acidity of the gastric juices. The most common clinical setting for gastric candidiasis is in patients with neoplastic disease. However, there are some case reports suggesting an increase in the prevalence of gastric candidiasis after gastric ulcer therapy with surgery or acid suppressive agents. Delayed gastric emptying, increased intragastric pH, and reflux of the duodenal contents into the stomach are factors indicative of the pathophysiology of gastric candidiasis after gastric surgery. We encountered a case of aggravated esophageal candidiasis and the formation of a gastric yeast bezoar following a gastric outlet obstruction due to a duodenal stenosis. We herein report this case along with an overview of the relevant literature. (Korean J Gastrointest Endosc 2007;34:205209)
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